ABSTRACT
This chapter looks at a range of possible origins of dyslexia in the hope of 'busting a few myths' and comparing serious research threads. It describes number of differing theoretical perspectives for the aetiology of dyslexia and current research strands. In the single gene model, there would be one dominant gene for dyslexia, which only one of the parents would need to transmit to their offspring for it to be expressed. When the neuroscientist Glen Rosen at Beth Israel induced a small lesion in the auditory cortex of these mice, neuronal abnormalities formed in the thalamus, similar to those found earlier in the dyslexic brain. C. Singleton and L. Henderson believe that visual stress could also be attributed to magnocellular deficits, or cortical hyperexcitability, which is a condition that possibly affects up to 20% of all children and 65% of children identified with dyslexia.
