ABSTRACT
Lesions of the superior parietal lobule are associated with impaired manipulative hand movements. The tight connections of the intraparietal sulcus with the premotor areas could explain the marked deficits in joint kinematic and dynamics occurring in apractic patients with lesions of this part of the parietal lobe. Damage to the posterior parietal cortex may cause the deficit of ideomotor limb apraxia. The observations indicated defects of visuo-kinaesthetic motor representation of learned movements that are stored in the dominant parietal lobe, or alternatively a separation of these representations from premotor or motor areas. Florida Apraxia Screening Test and modified Aachener Apraxia Test revealed signs of an ideomotor apraxia in two patients with left parietal damage and one with a right parietal lesion. The patient with a right parietal lesion without apraxia showed some preshaping and his maximum aperture was moderately increased and delayed.
