ABSTRACT
Panglial Syncytium .......................................................................................209 8.5 The Panglial Syncytium ...............................................................................209 8.6 NaV1 Channels at Nodes of Ranvier; KV1 Channels
Only at Juxtaparanodes and Internodes........................................................ 212 8.7 Oligodendrocytes Form Myelin in the CNS ................................................. 213 8.8 How Does Axonally-Released K+ Enter Myelin? ......................................... 216 8.9 Rethinking the Proposed Roles of Myelin in
Saltatory Conduction .................................................................................... 217 8.10 K+ Fluxes during Daily Turnover of Cerebrospinal Fluid
(CSF) Compared with K+ Ef¢uxes during High-Frequency Axonal Activity, with Relevance to Seizure Activity ................................... 218
8.11 Proposed Ultrastructural Pathways for K+ from Axon to Myelin Cytoplasm ......................................................................................... 219
8.12 Oligodendrocytes Myelinate Multiple Axons in the CNS, Thus Serving as a Distribution “Hub” ..........................................................220
8.13 Gap Junctions and Connexins of the Panglial Syncytium ............................ 221 8.14 Diseases of Panglial K+ and Water Transport Cause
