ABSTRACT
There are other numerous examples about the impacts of race, sex, and life style on the expression of genes. For this reason, the study of genetic and environmental interactions that modulate an individual’s responsiveness to medications has become one of the most vigorous areas in pharmacogenomic and toxicogenomic research (Borlak, 2005). Because of different genes they carry, and because of different environments in which these genes can be expressed, some people are more responsive to a drug than others. Thus, by identifying a network of interactions between the genes and enviroments, many fundamental questions in health sciences can be better addressed. These questions include how disease occurs and can be predicted, how potential environmental hazards and exposed individuals can be identified, and, in the ultimate, how disease can be prevented.
