ABSTRACT
The beneficial effects of probiotics arise from the interactions between the probiotic
bacteria, the host tissue, and the pathogens. Competitive exclusion of pathogens and
immuno-modulation may involve interaction between the adhesins on the bacterial surface
and the adhesion sites (in hydrophobic interaction) or specific receptors (in ligand-receptor
interaction) on the host cells.[1] This interaction is a dynamic process, and appropriate
interpretation of the kinetics would yield useful insight into the adhesion mechanisms
for the development of strategies to maximize probiotic effects for specific applications.
II. MATHEMATICAL MODELING
