ABSTRACT
Arteriovenous malformations (AVMs) are lesions composed of arteries and veins without intervening capillary beds. A high flow profile leads to vascular recruitment and arterialization of venous structures (1). It is believed that these lesions are most often congenital, arising at approximately the third week of gestation. At that point, an arrest in development results in the formation of direct arteriolar to venous communications without an intervening capillary bed (2).
