chapter  8
136 Pages


Throughout the four volumes of Chemical Sensitivity and the four volumes in the Mechanisms of Chronic Degenerative Disease and Chemical Sensitivity, pollutant exposure has been shown to have wide-ranging consequences. While some individuals appear to remain unaffected or have signicant time delays between an exposure, or set of exposures, and development of various sensitivities, illnesses, or end-organ diseases, others may experience immediate damage that ranges from insidious to grossly obvious effects (Chemical Sensitivity, Vol. 3, Chapter 28, p. 1935). Both immediate and delayed damage occurs in the sperm, ovum, placenta, fetus, the newborn, and the child. Epigenetics are rampant. Obvious reactions to pollutant exposure, such as asthma, eczema, or feeding problems, manifest immediately, while others, such as vasculitis, brain dysfunction, or arthritis, are delayed. In any of these circumstances, the damage that results may involve any individual or multiple organs and/or systems throughout the body. Following exposure to incitants, the patient with chemical sensitivity experiences the switching phenomenon, and normal body functions become dysfunctional. The epigenetic mechanism dominates genetics in most of this dysfunction. Chronic degenerative disease and both acute and chronic hypersensitivity occur during and after health deteriorates, before xednamed, end-stage, end-organ diseases occur. If the clinician can dene and eliminate or neutralize the environmental exposures, the child then can become well and with vigilance stay well.