ABSTRACT
Damage to the urethral sphincter and/or to mechanisms pertaining to its function is believed to be determinants in the pathogenesis of urinary stress incontinence. Undoubtedly, genetic and lifestyle factors that determine connective tissue strength and function are relevant in the etiology of stress incontinence. Pregnancy, childbirth, and in particular vaginal delivery are causative risk factors [1,2], and the condition may often present for the first time during pregnancy or postpartum.
