ABSTRACT
Myocardial ischemic syndromes pose a major medical problem and a significant economic health care concern. Reperfusion, although used in the clinical arena as essential to the survival of acutely ischemic heart muscle carries with it the risk of reperfusion injury. Therefore the salvage of additional myocardium is highly desirable.
TABLE OF CONTENTS
chapter Chapter 6|11 pages
Hsp72 in the Regulation of TNF-a Production:
Mechanistic Implication of Protection Against Postishemic Myocardial Dysfunction
chapter Chapter 9|13 pages
Stress Proteins in Myocardial Protection:
Culture Shock Protein, Heme Oxygenase-1 (Hsp32), Induced by Sublethal Stresses Protects the Heart Against Oxidative Stress
