ABSTRACT
Overexpression of a range of TaSnRK2s, including TaSnRK2.3, TaSnRK2.4, TaSnRK2.7, TaSnRK2.8 and W55a (a member with extremely high similarity to the formerly reported PKABA1) lead to acquired tolerance of multiple-abiotic stress in transgenic Arabidopsis. This can be attributed to a series of improved physiological traits and altered expressions of abiotic stress-responsive genes (Mao et al. 2010; Tian et al. 2013; Xu et al. 2009; Zhang et al. 2010, 2011). TaSK5, encoding an abiotic stress-inducible GSK3/shaggy-like kinase, is constitutively induced by abiotic stress and confers enhanced tolerances to salt and drought in Arabidopsis (Christov et al. 2014). TaABC1 encodes a protein kinase ABC1 (activity of bc (1) complex). Overexpression of TaABC1 confers improved tolerances to drought, salt and cold stresses (Wang et al. 2011). TaPI4KII-g (phosphatidylinositol (PI) 4-kinases PI4Ks) participates in the regulation of recruitment and the activity of signalling proteins in cell membranes, promoting root growth and altering the expression patterns of stress-related genes. Hetero-expression of TaPI4KII-g leads to pronounced tolerance to drought stress in Arabidopsis (Liu et al. 2013).
