ABSTRACT
INTRODUCTION Chronic ischemic heart disease (IHD) is accompanied by an increased risk of both tachyarrhythmias and bradyarrhythmias. Myocardial ischemia leads to changes in impulse conduction velocity and influences the refractory periods. The development of myocardial infarcts (MI) creates zones of nonconducting tissue surrounded by border-zones with changed electrophysiological properties, thus making the settings for re-entry loops.
