ABSTRACT
Although the etiology and pathogenesis of multiple sclerosis (MS) may still be considered by many to be ‘unknown’, the working hypothesis of most investigators is that MS is a cell-mediated autoimmune disease directed against central nervous system (CNS) myelin and is related in some way to a viral infection.1 Furthermore, with recent advances in our understanding of the immune system and the demonstration that certain treatments can affect the clinical course of MS, it is now possible to approach the disease in more positive terms.
