ABSTRACT
Ocular tissues, like those of other organs, exhibit well-defined morphological reactions to local trauma and insult in the form of hyperemia, vasodilatation, increased permeability of blood vessels and edema. Cystoid macular edema (CME) following cataract surgery is one such manifestation, and it can result in either temporary and rarely a permanent reduction of visual acuity. Although its etiology remains obscure, its relationship to the details of the surgical procedures and their complications are becoming clearer. Likewise, more insight into the phenomenon is also being imparted by the agents being used in the preventive or curative line of management in CME.
