ABSTRACT
Despite the extensive and sustained use of dinitroaniline herbicides, only a few examples of resistance have been reported. This chapter describes the laboratory and field studies on the resistant biotypes that have arisen, and discusses the mechanisms by which these weed biotypes and various crops resist these herbicides. The complex segregation indicates that several genes are responsible for resistance, although the numbers of offspring were too small to substantiate any genetic model. The number of reports documenting resistance to dinitroanilines is relatively small given the long-term and widespread use of these herbicides. Those resistant biotypes that have been selected offer an intriguing view as to what perturbations to the cytoskeletal proteins can occur and their consequences. The discovery of weeds resistant to dinitroaniline herbicides has prompted studies to determine cross resistance to compounds structurally dissimilar from dinitroaniline herbicides, but which also disrupt mitosis.
