ABSTRACT
Within the molecular mechanisms of cancer, reactive oxygen species play a fundamental role; they are capable of acting on vital processes such as angiogenesis and apoptosis [1].
Angiogenesis and the inhibition of apoptosis are two processes that favor tumor development. It is known that factors that promote angiogenesis are activated by oxidative stress; for example, when endothelial cells are exposed for 30 minutes to hydrogen peroxide (H2O2), a stimulation of angiogenesis is observed. It has now been shown that reactive oxygen species (ROS) such as the superoxide anion radical and H2O2 are involved in neovascularization [1].
