ABSTRACT
The proposed relationship between intrapartum asphyxia and cerebral palsy is based on two observations: asphyxia in the animal model can produce brain pathology consistent with cerebral palsy in the human; some human newborns that are subsequently shown to have cerebral palsy were asphyxiated at birth. The preponderance of evidence from the medical literature supports the point of view that most cerebral palsy is not related to intrapartum events. Perhaps the most convincing reason for identifying the prenatal period rather than the intrapartum period as the time frame for the development of mocan is found inst cases of cerebral palsy studies which have attempted to study directly the effect of modern obstetric interventions. The brain lesion produces a myocardial effect or a change in cardiac rhythm which results in cardiac decompensation and marked changes in the Electronic Fetal Monitoring pattern consistent with fetal asphyxia.
