ABSTRACT
Lupus erythematosus (LE) is a chronic, autoimmune, multisystem disease that may trigger inflammation in several organs, with skin representing one of the most commonly involved sites, either alone or in combination with other organs’ involvement.1 Cutaneous manifestations of LE are often induced or aggravated by ultraviolet light and can be divided into “specific” and “nonspecific” based on histopathological findings, with the former being typically characterized by a lichenoid tissue reaction.1 LE-specific skin manifestations can be subdivided into acute cutaneous LE (ACLE), subacute cutaneous LE (SCLE), and chronic cutaneous LE (CCLE).1
