Acne vulgaris, not unlike other abnormalities of the skin, has both intrinsic and extrinsic factors that influence the pathology. Traditionally cited intrinsic factors include (1) excessive sebum, (2) hyperkeratinization of the sebaceous follicle, and (3) rupture of the follicular epithelium, which leads to the appearance of the inflammatory lesions. The microorganism Propionibacterium acnes has also been said to play a role in the inflammatory phase of this disease (1).