ABSTRACT

The human diet, as well as containing a wide array of carcinogens (Lutz, 1992) also contains a similarly wide array of anticarcinogens. These latter are detected in model systems by their ability to inhibit carcinogenesis and so, of necessity, their detailed study has had to await a detailed understanding of the test systems. Nevertheless, despite early work by, for example, Crabtree (1947) and later interest from such leaders as Ames (1983) there has been relatively little interest in experimental anticarcinogenesis.