ABSTRACT
Nevertheless, angiographically visible vessel dissection without reduction of flow is associated with a 6.5-fold increase in the incidence of acute or subacute closure.10 On the other hand, plaque dissection is the most significant mechanism of lumen enlargement after balloon dilatation, as shown by ultrasound11-13 and angioscopy14
studies, and it is likely that plaque fracture is a prerequisite for achieving effective and persistent plaque compression and redistribution and lumen enlargement. Ultrasound studies suggest that temporary wall stretch in concentric lesions or in eccentric lesions in vessels with a disease-free arc without plaque disruption and dissection has a smaller lumen gain due to a significant immediate elastic recoil after dilatation.13
