ABSTRACT
The goals of this treatment are minimization of brain ischemic injury caused by the
embolus and prevention of acute recurrent embolism. Strategies for accomplishing this
fall into four broad categories: (i) reperfusion of the brain region rendered ischemic by
the embolus, (ii) acute anticoagulation to prevent propagation and further embolization
of thromboemboli, (iii) making the brain more resistant to ischemia, allowing survival
of nerve cells despite ischemia, and (iv) managing complications of embolic infarction,
such as brain edema and brain hemorrhage.