ABSTRACT
Restenosis after peripheral artery intervention represents a problem of different magnitude depending upon the target vessel type of location. Rates range from less than 5% in carotid arteries to as high as 53% in infrainguinal locations. These extreme differences are explained by numerous factors including: muscular versus elastic type of arteries (as discussed previously), target vessel diameter, plaque burden, and lesion morphology at presentation. Due to the major differences in restenosis, incidence, and modulating factors depending on the anatomic bed, the most important different anatomic vascular regions will be discussed separately.
