Anorexia nervosa (AN) is a devastating illness in which behavior is profoundly narrow, rigid, and seemingly disconnected from somatic experience. This behavioral profile, when combined with the self-imposed starvation pathognomonic of the disorder, contributes to the designation of eating disorders as one of the 10 leading causes of disability among women (Mathers, Vos, Stevenson, & Begg, 2000; Striegel-Moore & Bulik, 2007). While advances have been made in the management of adolescent AN (Keel & Haedt, 2008; Lock & Fitzpatrick, 2009); a significant minority fail to directly benefit from treatment (Eisler, Simic, Russell, & Dare, 2007), comorbid psychopathology persists (Herpertz-Dahlmann et al., 2001), and crossover to other forms of eating disorders is common (Eddy et al., 2008; Tozzi et al., 2005). Moreover, recommended treatments for adults with AN remain elusive (Berkman, Lohr, & Bulik, 2007; Bulik, Berkman, Brownley, Sedway, & Lohr, 2007; Wilson, Grilo, & Vitousek, 2007).