ABSTRACT
As the preceding chapters illustrate, our understanding and management of LAH and ACS as potentially life-threatening concerns in the critically ill have evolved tremendously since their rediscovery. Significant strides have been made in a relatively brief period of time in both our knowledge of the pathophysiology involved and the interventions necessary to improve patient outcome. We now recognize that the etiology o f LAH and ACS is commonly multi-factorial, that there are several different forms of both LAH and ACS, and that early detection and management significantly improves survival.
