ABSTRACT
Cushing’s syndrome typically manifests with various neuropsychiatric features (e.g., depression), classically in a patient with a ‘Cushingoid’ habitus consisting of ‘moon’ facies, truncal obesity, and violaceous abdominal striae (Orth 1995). This syndrome occurs secondary to the effects of sustained hypercortisolemia, which in turn may be due to either administration of exogenous steroids (e.g., prednisone) or endogenous overproduction of steroids.
