ABSTRACT
This chapter discusses a variety of verbal and motor behaviours that are often encountered in patients with Transcortical Aphasias (TA). It considers the status of linguistic and nonlinguistic prosody in TA, by describing recent evidence based on clinical and acoustic studies. EP is subsequently inhibited in parallel with the progression of language and gesture development. It may reappear, for instance, during the learning of a second language where normal individuals use noncompulsive verbalisation and completion of openended sentences to reinforce auditory comprehension during the learning processes. On interpreting the pathophysiology of language disturbances, these authors suggested that the thalamic lesions caused a loss of complex language functions due to insufficient activation of “superior” cortical language areas, but left the more automatic speech processes relatively spared. Ross evaluated the production of affective prosody in various clinical types of aphasia, and compared the patient’s performance on a quantitative verbal-articulatory task with those obtained from a group of right brain-damaged patients with aprosodia.
