ABSTRACT
Background Diabetic foot syndrome is perhaps one of the most important complications of diabetes mellitus. The prevalence of diabetic foot ulcers ranges between 4-10%, while the lifetime incidence rates may be as high as 25% (1). A number of factors like the peripheral vascular disease, changes in foot architecture, peripheral sensory neuropathy and the plantar pressure are considered to be the prime etiological factors for the development of ulcers (2,3). Studies in the past have demonstrated that, the capillary fl ow is increased rather than decreased in the diabetic neuropathic foot and the high pressure areas are presumed to be associated with increased basal skin blood fl ow as compared to low pressure areas (4-6). Currently it is believed that the development of plantar pressure ulcers is associated with high amount of pressure exerted on certain regions of the foot (7-10).
